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對新冠病毒的簡單毒性理论解釋
疫苗注射確實 记者综述：关于朊病毒的发现并获得诺贝尔医学奖历史典故
please translate into english
A simple theoretical explanation of the toxicity of coronavirus injections
Vaccine Injection is Real Reporter’s Summary: Historical Allusions about the Discovery of Prions and Winning the Nobel Prize in Medicine

利迪亞·安吉洛娃博士

2023年5月26日10:45

審查的

真正的前景

訂閱

似乎有太多的人仍然不瞭解所謂的mRNA疫苗的基礎知識，它們根本不是疫苗，而是我所說的基因產物。

以下是對這些產品的工作的最基本解釋。

记者综述：关于朊病毒的发现并获得诺贝尔医学奖历史典故。

Dr. Ryan Cole: "Human cells are meant to make human proteins, not to make toxins." "To inject a gene-based product into the body and make your cells the factory for a toxin is psychologically insane."

https://open.substack.com/pub/angelovalidiya/p/simple-explanation-of-what-covid

My simple explanation..Removed the paywall which is the right thing to do.

Please consider supporting my work. 瑞安·科尔博士：“人类细胞的目的是制造人类蛋白质，而不是制造毒素。” “将基于基因的产品注入体内并使你的细胞成为毒素工厂在心理上是疯狂的。”

我的简单解释..删除了付费专区，这是正确的做法。

请考虑支持我的工作。 https://substack.com/@angelovalidiya/note/c-195891365

It appears that far too many people still do not understand the fundamentals of so-called mRNA vaccines, which are not vaccines at all and are instead what I refer to as gene products. The following is the most basic explanation of what these products do.
Think of the cell as a little factory 🏭. Imagine human body as a tiny, remote town with many factories like this🌇 (cells). Each factory has specific machines that produce specific products, on which the town is dependent.
Most of the factories in town are eventually ordered to produce a hazardous product called spike protein (COVID vaccines). The factories have never made anything like this before.
Now the factories must create the new product while continuing to manufacture the old.
There were no additional supplies delivered to the factories. Everything is the same, except for the extra work.
The factories do not have instructions on how long the product must be manufactured.
What happens next?
Since each factory uses the same machines and supplies, it cannot produce all products. Some products are no longer manufactured or are manufactured in smaller quantities.
Given that the extra product is brand new, the factory may not always produce it at the highest quality (prions).
Since the factory does not have directions on when to stop producing this product, so it continues to produce it while making fewer and fewer of the other products.
The factory (cell) eventually runs out of resources and shuts down (cell death) 💀.
What happens in the town?
The town in which the factories are located is dependent on the products that these factories provide.
The town does not receive the products it requires, but instead receives wasteful products. At some point, the town is submerged by wasteful products with detrimental effects. The town (human body) shuts down 💀.
Again, the factory is the cell that produces spike protein after injection with so-called vaccines. The town is the inoculated person's body. Many factories in town, i.e. cells in the body, produce toxic proteins on a continuous basis while being unable to produce other necessary molecules. Cells and body will eventually shut down. Because humans are complex organisms and the vials do not contain the same amount of product, bodies will react differently, resulting in different adverse reactions, but the end result will be always the same - shutting down or death. 💀. 看来还有太多人不了解所谓的mRNA疫苗的基本原理，它们根本不是疫苗，而是我所说的基因产品。 以下是这些产品功能的最基本解释。
将细胞视为一个小工厂🏭。 将人体想象成一个偏远的小镇，有许多像这样的工厂🌇（细胞）。 每个工厂都有生产特定产品的特定机器，该镇依赖这些机器。
镇上的大多数工厂最终都被命令生产一种名为刺突蛋白（新冠疫苗）的危险产品。 工厂以前从未生产过这样的东西。
现在工厂必须在继续生产旧产品的同时创造新产品。
没有额外的物资运送到工厂。 除了额外的工作之外，一切都一样。
工厂没有关于产品必须生产多长时间的说明。
接下来会发生什么？
由于每个工厂都使用相同的机器和用品，因此无法生产所有产品。 有些产品已不再生产或生产数量较少。
鉴于额外的产品是全新的，工厂可能并不总是以最高质量（朊病毒）生产它。
由于工厂没有关于何时停止生产该产品的指示，因此它继续生产该产品，而生产的其他产品越来越少。
工厂（细胞）最终耗尽资源并关闭（细胞死亡）💀。
镇上会发生什么？
工厂所在的城镇取决于这些工厂提供的产品。
该镇没有收到所需的产品，而是收到了浪费的产品。 在某些时候，该镇被具有有害影响的浪费产品淹没。 城镇（人体）关闭💀。
同样，工厂是注射所谓疫苗后产生刺突蛋白的细胞。 城镇就是接种者的身体。 镇上的许多工厂，即体内的细胞，不断生产有毒蛋白质，但却无法生产其他必需的分子。 细胞和身体最终都会关闭。 由于人类是复杂的有机体，并且小瓶中所含的产品量不同，人体的反应会有所不同，从而导致不同的不良反应，但最终结果总是相同的——停工或死亡。 💀。

I'm doing everything I can to assist those who have received the COVID-19 vaccines. Here's what I've got for them.

我正在盡我所能幫助那些接種了新冠肺炎疫苗的人。 這是我為他們準備的。

我免費製作了它，沒有從上述產品中獲利。 我的自然療法系列也將使他們和其他人受益。

I made it for free and do not profit from the products mentioned. My Natural Remedy Series would also benefit them and everyone else.

網址：https://angelovalidiya.substack.com/s/natural-remedy-系列

我知道我所做的對人們有幫助，但我仍然失業。 沒有人伸手來幫助我。 請考慮透過我的部落格

……

Reporter's Summary：

Prions: Discovery and Nobel Prize in Physiology or Medicine

Prions are a class of infectious agents made up of proteins. What sets them apart is that they do not rely on nucleic acids (such as DNA or RNA) for transmission or replication. Instead, they cause disease by altering the three-dimensional structure of host proteins. This discovery challenged the traditional biological belief that infectious agents must depend on nucleic acids to propagate, thus drawing significant attention within the medical community.

Discovery of Prions and the Nobel Prize in Medicine

The discovery of prions is credited to American scientist Stanley B. Prusiner. In 1982, Prusiner proposed that certain neurodegenerative diseases, such as Bovine Spongiform Encephalopathy (BSE, commonly known as “mad cow disease”) and Creutzfeldt-Jakob Disease (CJD), are caused by a special type of protein, rather than a virus or bacterium. This protein causes damage to the nervous system by altering the structure of normal proteins.

Prusiner’s discovery was highly controversial at the time, as traditional biological theory held that all infectious agents required genetic material (such as DNA or RNA) to spread. However, Prusiner was convinced that certain proteins could, in the absence of nucleic acids, spread disease by altering the structure and function of nearby proteins.

Stanley B. Prusiner’s Contribution and the Nobel Prize

Prusiner’s work faced significant skepticism, but he persisted in advancing his research. Eventually, his theory gained support from experimental data, particularly through his studies on the mechanisms of diseases like BSE and CJD. He was able to demonstrate that these diseases are indeed caused by abnormal proteins—prions.

In 1997, Prusiner was awarded the Nobel Prize in Physiology or Medicine for his discovery of prions. This honor marked the medical community’s acceptance of “non-traditional infectious agents” and paved the way for further research and exploration of prions.

The Impact of Prions

Prusiner’s research not only unveiled a completely new mechanism of infection, but it also deepened the medical community’s understanding of diseases linked to protein misfolding. Neurodegenerative diseases caused by prions, such as Alzheimer’s disease and Parkinson’s disease, may be related to the misfolding and accumulation of proteins. Therefore, the discovery of prions not only aids in the diagnosis of these diseases but also offers new avenues for developing therapeutic strategies.

Additionally, the study of prions has had significant implications for the agricultural industry. Following the outbreak of mad cow disease, scientists began to place greater emphasis on prion contamination in animal products. This led to the implementation of stricter control measures to prevent the transmission of such fatal diseases to humans via the food chain.

Conclusion

Through his perseverance and innovation, Prusiner changed our understanding of infectious pathogens, leading the medical community to recognize that proteins themselves can be pathogenic agents. His work expanded the field of molecular biology and provided new insights into the fields of neuroscience, immunology, and disease prevention.

However, criminals with anti-human intent have used it to create horrifying biological weapons.

记者综述：关于朊病毒的发现并获得诺贝尔医学奖历史典故。

朊病毒（Prion）是一类由蛋白质构成的感染性因子，其独特之处在于它们并不依赖于核酸（如DNA或RNA）来传播或复制，而是通过改变宿主蛋白质的三维结构来引发疾病。这一发现打破了传统生物学中关于感染性物质必须依赖核酸的常规认知，因此在医学界引起了广泛关注。

朊病毒的发现与诺贝尔医学奖

朊病毒的发现归功于美国科学家 斯坦利·B·普鲁西纳（Stanley B. Prusiner）。普鲁西纳在1982年提出，某些神经退行性疾病，如疯牛病（Bovine Spongiform Encephalopathy，BSE）和克雅病（Creutzfeldt-Jakob Disease，CJD）等，是由一种特殊的蛋白质引发的，而非由病毒或细菌引起。这种蛋白质通过改变正常蛋白质的结构，导致神经系统的损伤。

普鲁西纳的这一发现受到了极大的争议，因为传统的生物学理论认为，所有感染性物质都需要依赖遗传物质（如DNA或RNA）才能传播。然而，普鲁西纳坚信，某些蛋白质能够在没有核酸的情况下，通过改变周围蛋白质的结构和功能，传播疾病。

斯坦利·B·普鲁西纳的贡献与诺贝尔奖

普鲁西纳的工作在当时遭到了广泛的质疑，但他依然坚韧不拔地推进了自己的研究。最终，他的理论得到了实验数据的支持，尤其是他通过研究疯牛病（BSE）、克雅病（CJD）等疾病的致病机制，证明了某些疾病确实是由异常蛋白质——即朊病毒——引起的。

1997年，普鲁西纳因其关于朊病毒的发现荣获诺贝尔生理学或医学奖。这一获奖标志着医学界对“非传统感染因子”的接受，开启了对朊病毒的进一步研究和探索。

朊病毒的影响

普鲁西纳的研究不仅揭示了一个全新的感染机制，也促使医学界对与蛋白质折叠错误相关的疾病有了更深刻的理解。朊病毒所引发的神经退行性疾病，如阿尔茨海默病、帕金森病等，可能与蛋白质错误折叠和积聚有关。因此，朊病毒的发现不仅有助于疾病的诊断，也为开发新的治疗方法提供了新的思路。

此外，朊病毒的研究也对农业产业产生了重要影响，特别是在疯牛病的爆发后，科学家们更加关注动物产品中的朊病毒感染，采取了更加严格的防控措施，以避免人类通过食物链传播此类致命的疾病。

结语

普鲁西纳通过他的坚韧与创新，改变了我们对感染性病原体的理解，使医学界认识到蛋白质本身也可以是致病因子。他的工作不仅扩展了分子生物学的研究领域，也为后来的神经科学、免疫学以及疾病防控提供了新的思路。

然而！反人类不法之徒用以制造骇人听闻的生物武器。