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A National Institutes for Health (NIH) spokesperson is disputing a nonprofit watchdog group’s claim that the agency “deleted” genetic sequencing data on the CCP virus from a Chinese lab, but the same official acknowledged the data was “suppressed.”
“The headline says the sequences were deleted which is inaccurate. They were not deleted. This is a really important point, and I’ve highlighted what did happen from what we provided to you earlier this week,” NIH Media Branch Chief Amanda Fine told The Epoch Times in a March 31 email.
Fine was referring to a March 29 Epoch Times story headlined “NIH Deleted Info Received From Wuhan Lab on CCP Virus Genetic Sequencing, Watchdog’s FOIA Finds.” The information Fine referenced as having been provided to The Epoch Times by NIH earlier in the week was included in the published story:
“’In June 2020, in response to a request by the same [Wuhan] researcher, National Center for Biotechnology [NCBI] gave the sequence data the status of “withdrawn,” which removes sequencing data from all public means of access but does not delete them.
“‘NCBI subsequently reassigned the status of the sequence data to “suppressed,” which means that sequence data are removed from the search process but can be directly found by accession number. This action to reassign the data was identified as part of NLM’s ongoing review into the matter. We are working to make more information available,’ the spokesperson said.”
The biotechnology center, which is part of the institute’s National Library of Medicine (NLM), is the U.S. component of the International Nucleotide Sequence Database Collaboration.
The Epoch Times story was prompted by a report published on March 29 by Empower Oversight Whistleblowers and Research (EO) that was based on Freedom of Information Act (FOIA) responses the group received from the institute.
“On June 5, 2020, a Wuhan University researcher requested that NIH retract the researcher’s submission of BioProject ID PRJNA637497 because of error. The Wuhan researcher explained ‘I’m sorry for my wrong submitting,'” Empower Oversight said in a statement (pdf) on March 29.
“BioProject ID PRJNA637497 is also referred to as Submission-ID SUB7554642. Three days later, on June 8th, the NIH declined the researcher’s request, advising that it prefers to edit or replace, as opposed to delete, sequences submitted to the SRA,” EO reported. SRA refers to the Sequence Read Archive (SRA) data resource made available by NCBI, and it “stores raw sequencing data.”
“But then, on June 16, 2020, NIH officials reversed themselves and deleted the genetic sequencing data, as requested by the Wuhan researcher. That researcher was quoted by EO as explaining to NIH: ‘Recently, I found that it’s hard to visit my submitted SRA data, and it would also be very difficult for me to update the data. I have submitted an updated version of this SRA data to another website, so I want to withdraw the old one at NCBI in order to avoid the data version issue.’
“After some discussion about what would be deleted, the NIH concluded the discussion by reassuring the Wuhan researcher that it ‘had withdrawn everything.’”
Asked for a response to Fine’s claim the information was not deleted, EO Founder and President Jason Foster told The Epoch Times that NIH’s actions ensure the CCP virus genetic sequencing info is only available to the few individuals possessing its “accession number,” which effectively deletes the data from open access and research.
“NIH documents released with Empower Oversight’s report demonstrate that the sequencing data was deleted from public view by the NIH at the request of the Wuhan researcher,” Foster said.
Ah yes, Mr. Vandervelde, but that’s not the point is it.
By Dr. Michael Yeadon via Doctors for Covid Ethics
The Covid Lies – Doctors for COVID Ethics (doctors4covidethics.org)
In this comprehensive review, Dr. Yeadon argues that all the main narratives about SARS-CoV-2 and imposed “measures” are lies.
In the first part of the article (The Covid Lies), Dr. Yeadon counters the 12 widespread Covid narratives with the following arguments:
The infection fatality rate of SARS-CoV-2 is 0.1 – 0.3%, which is not significantly different from some seasonal influenza epidemics.
Based on the peer-reviewed articles, at least 30 to 50% of the population has prior cross-immunity.
SARS-CoV-2 does discriminate. “The lethality of this virus, as is common with respiratory viruses, is 1000X less in young, healthy people than in elderly people with multiple comorbidities.”
Asymptomatic transmission is the “central conceptual deceit” used to “underscore almost every intrusion: masking, mass testing, lockdowns, border restrictions, school closures, even vaccine passports.”
PCR test is “the central operational deceit.”
Neither cloth nor surgical masks prevent respiratory virus transmission.
Lockdown is “epidemiologically irrelevant” and never works. “Only “stay home if you’re sick” works.“
“Covid-19 is the most treatable respiratory viral illness ever”. Safe and effective early treatments are available.
Based on the peer-reviewed articles, very few clinically significant reinfections of SARS-Cov-2 have ever been confirmed.
SARS-CoV-2 mutates slowly, and no variant is even close to escaping naturally-acquired immunity. However, there is the possibility that the so-called vaccines prevent the establishment of immune memory, leading to the repeated infections, which would be a form of acquired immune deficiency.
Safety is the top priority in a public health mass intervention, even more than effectiveness. “It was NEVER appropriate to attempt to “end the pandemic” with a novel technology vaccine.”
The four gene-based “vaccines” are toxic. The basic rules of selecting vaccine candidates are: 1) the agent has no inherent biological action (non-toxic); 2) the agent should be the genetically most stable part of the virus; 3) the agent should be most different from human proteins. Spike protein as the vaccine does not fit any of the above criteria.
these are the people whose covid responses the whole world threw away 100 years of evidence based pandemic guidelines to copy and cheer-lead for in 2020.
still feeling good about that decision?
UCSD Is Sending Positive Tested Students To Quarantine Facilites
This is happening now
https://returntolearn.ucsd.edu/campus-guidelines/latest-updates/index.html?source=patrick.net
https://returntolearn.ucsd.edu/campus-guidelines/testing-and-screening/student-screening-and-testing/index.html?source=patrick.net#quarantine-isolation-housing
Fuck Hawaii!
Anyone wants to risk a bet on the eventual number of sick people? Dead people?
SARS-2 surges only in the winter, goes endemic after two waves, is impervious to vaccination, and has become harmless with Omicron
A brief look at the Corona pandemic in Europe from the perspective of excess mortality
A lot of the Corona data we’re fed is essentially meaningless propaganda, and it has obscured crucial patterns. Here, I want to look at the only metric that really matters, namely excess mortality, to make some basic points about what has happened to us since 2020, and what is happening now.
To date, the pandemicists have counted five or six waves of infection. If you ignore the case statistics, though, and look at nothing but European excess mortality, you see a totally different picture. Corona only has one deadly season a year, namely the winter, and no European country has seen more than two winter mortality spikes. ...
It’s definitely Omicron, and not the vaccines, that stopped the deaths. The same pattern exists in Austria, which is slightly less heavily vaccinated than Germany; and also in Hungary and Slovenia, which are substantially less vaccinated. ...
In Europe, Corona kills people for two seasons, and then it becomes a nothingburger, no worse than seasonal influenza in a bad year. You see exactly the same picture in England – two mortality waves, and then it’s over...
In most countries, the two-wave mortality surge was over by the time the vaccines were rolled out; in central Europe, it was still raging, but it was Omicron and not the vaccines that stopped the deaths.
has become harmless with Omicron
Towards a General Theory of Corona and Why No Interventions Ever Seem to Work As Intended
Why have lockdowns and other containment measures proven so generally ineffective against SARS-2? What causes waves of infection to begin, and why do they collapse long before all the susceptible have been infected? What is the significance of asymptomatic infection? Why do the vaccines seem to boost infections for a few weeks after the first and third doses? Why did the SARS-2 spike protein begin to experience heavy selection pressure in the latter half of 2020, giving rise to variants like Alpha and Delta? How did these variants manage so completely to replace prior lineages?
Increasingly, I wonder whether all of these questions aren’t just facets of the same phenomenon – one that was first described by R. Edgar Hope Simpson in his fascinating book on The Transmission of Epidemic Influenza (New York, 1992). As I said on Twitter, Hope-Simpson is one of the only people to have attempted what might be called a zoology of viruses. That is to say, he tried to analyse and explain how influenza interacted with its human hosts more broadly, and not just how it infected cells and responded to antibodies. This is precisely where modern epidemiology and virology are most deficient, with extremely simplistic understandings of how viruses behave across the population. ...
Hope-Simpson therefore concluded that influenza transmission was a far more subtle matter, than sick people infecting the healthy.
He proposed instead a two-phase process:
1) Acute infection, lasting a week or two, during which influenza is only transmitted very rarely.
2) Latency, whereby a subset of recovered people fail to clear the virus completely, and instead become asymptomatic carriers for many months, until seasonal factors correlated with latitude cause them to become infectious once again, generally without any noticeable recurrence of illness.
Alpha might have spread faster than wild-type SARS-2, but how did it actually succeed in wiping out these wild-type strains? Hope-Simpson calls this the “disappearing act,” as it is also observed with successive strains of influenza, and is very hard to account for.
Hope-Simpson finds the beginning of an answer in old influenza A antibody experiments:
During the great influenza A epidemic of the 1950–51 season, Isaacs in London, England and independently Archetti and Horsfall in the United States made a seminal observation. Two minor variants of the A (H1N1) subtype, “Scandinavian” and “Liverpool,” were co-circulating in many parts of the world. In both laboratories it was discovered that if the Scandinavian strain were made to infect a fertilized chicken egg in the presence of Scandinavian antibody, the strain harvested a few days later would turn out to be Liverpool. Vice versa, an appropriate dose of homologous antibody induced Liverpool virus-infected eggs to yield a harvest of Scandinavian virus.
The findings have been repeated with other strains of influenza virus in many laboratories throughout the world. (p. 99)
Some influenza strains, in other words, emerge spontaneously, via mutation, as a response or solution to the antibodies generated against other strains. Thus Hope-Simpson held that selection for new influenza strains happened primarily in the latent carriers of the virus, after acute infection.
Over months, the virus that lurked in carrier tissues was able to train itself against their immune response. Various mutant strains arose, and when the carriers became infectious again, the most contagious of these mutants went on to infect other people as the new influenza strain. Because the set of optimal solutions was in every case limited, all the latent carriers infected with the prior strain ended up incubating the same set of successor strains.
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