Friday Hope: Black Seed Oil: Inhibiting Spike Binding, Inflammatory and Coagulation Responses and Enhancing p53 Expression Thymoquinone (TQ), the major component of Black Seed Oil, may be a useful adjunct therapy in preventing/treating the damaging effects of the Spike Protein. WALTER M CHESNUT MAY 2
星期五希望：黑色种子油：抑制尖峰结合，炎症和凝结反应并增强p53表达 黑籽油的主要成分胸醌（TQ）可能是防止/治疗峰值蛋白的破坏作用的有用辅助疗法。 Walter M Chesnut 5月2日
Friday Hope: Black Seed Oil: Inhibiting Spike Binding, Inflammatory and Coagulation Responses and Enhancing p53 Expression Thymoquinone (TQ), the major component of Black Seed Oil, may be a useful adjunct therapy in preventing/treating the damaging effects of the Spike Protein. WALTER M CHESNUT MAY 2
READ IN APP
Thymoquinone may prevent SARS- CoV- 2 virus receptor interaction by inhibition of the attachment of the viral spike glycoprotein to the Angiotensin Converting Enzyme 2 (ACE2) receptor and the priming of SARS- CoV- 2 virus through TMPRSS2 (transmembrane protease serine 2).
It is now coming to the attention of doctors worldwide that levels of the tumor suppressor p53 are declining alarmingly. Here is just one example:
Unfortunately, readers of this Substack know that this has been expected due to the previously discussed interaction of the Spike Protein with p53 and other tumor suppressors. I have written in a previous Friday Hope about some natural therapeutics which can enhance p53 expression. Today I would like to discuss one additional natural therapeutic which not only enhances p53 expression but also prevents Spike binding and ameliorates the inflammatory/coagulation responses it induces.
Black Seed Oil, or Black Cumin Oil, has a long and highly regarded history of being an important, multi-functional nutraceutical.
One spicy, medicinal herb is Nigella sativa L. (Ranunculaceae), also called black cumin or black seeds, which is famous for its culinary uses, and is historically precious in traditional medicine. Black cumin is native to a vast region of the eastern Mediterranean, northern Africa, the Indian subcontinent, and Southwest Asia, and is cultivated in many countries, including Egypt, Iran, Greece, Syria, Albania, Turkey, Saudi Arabia, India, and Pakistan. Being a panacea, black cumin, in the form of essential oil, paste, powder, and extract, has been indicated in traditional medicine for many diseases/conditions, such as asthma, bronchitis, rheumatism, headache, back pain, anorexia, amenorrhea, paralysis, inflammation, mental debility, eczema, and hypertension, to name a few [4]. These traditional uses of N. sativa seeds are largely attributed to their wide array of medicinal properties, including antioxidant, anti-inflammatory, immunomodulatory, anticancer, neuroprotective, antimicrobial, antihypertensive, cardioprotective, antidiabetic, gastroprotective, and nephroprotective and hepatoprotective properties [5]. Black cumin seed, particularly its essential oil, contains thymoquinone (TQ), thymohydroquinone, thymol, carvacrol, nigellidine, nigellicine, and α-hederin, which are mostly responsible for its pharmacological effects and therapeutic benefits [6]. The food value of black cumin, although less focused on in scientific literature, is by no means low, because it contains an adequate quantity of protein and fat, and an appreciable amount of essential fatty acids, amino acids, vitamins, and minerals [7]. Both active phytochemicals and the vital nutrients of black cumin contribute equally to the immunity and well-being of the human body, making this culinary herb a valuable source of nutraceuticals.
There are additional benefits to Black Seed Oil which are specifically related to SARS-CoV-2 and its Spike Protein. Black Seed Oil and its major component, Thymoquinone (TQ), is a promising therapeutic for COVID and Spike Protein injury/disease. Three of these benefits are very important: preventing the Spike from binding to our cells and quelling the inflammatory and coagulation responses that the Spike and the virus itself induces.
Compounds derived from N. sativa oil, in particular TQ, may be effective in inhibiting the ability of SARS- CoV- 2 to attach to host cell receptors and replicate within the cell.
Covid 19 is also associated with an aggressive inflammatory and hyperactive host immune response to the SARS- C0V- 2 virus which is commonly referred to as a ‘cytokine storm’.52 Proinflammatory cytokines/chemokines, including multiple interleukins and TNF- α, are elevated in Covid19 patients and contribute to worse patient outcomes.52 Nigella sativa and TQ may be potentially beneficial in downregulating this inflammatory process and thereby reducing the severity of acute respiratory distress syndrome.51,52 Nigella sativa and TQ also possess anticoagulant properties that may offer both as potential therapeutic natural products targeting the coagulopathy associated with life- threatening SARS- CoV- 2 (Covid 19) infection.
Furthermore, there is yet another property that TQ possesses, which may prove to be very beneficial for those exposed to the Spike Protein. Thymoquinone has the ability to enhance expression of the tumor suppressor p53, which the Spike Protein downregulates. It also inhibits the growth of colon cancer cells.
We investigated the effects of thymoquinone (TQ) against HCT-116 human colon cancer cells and attempted to identify its potential molecular mechanisms of action. We report that TQ inhibits the growth of colon cancer cells which was correlated with G1 phase arrest of the cell cycle. Furthermore, TUNEL staining and flow cytometry analysis indicate that TQ triggers apoptosis in a dose- and time-dependent manner. Apoptosis induction by TQ was associated with a 2.5-4.5-fold increase in mRNA expression of p53 and the downstream p53 target gene, p21WAF1. Simultaneously, we found a marked increase in p53 and p21WAF1 protein levels but a significant inhibition of anti-apoptotic Bcl-2 protein. Co-incubation with pifithrin-alpha (PFT-alpha), a specific inhibitor of p53, restored Bcl-2, p53 and p21WAF1 levels to the untreated control and suppressed TQ-induced cell cycle arrest and apoptosis. p53-null HCT-116 cells were less sensitive to TQ-induced growth arrest and apoptosis. These results indicate that TQ is antineoplastic and pro-apoptotic against colon cancer cell line HCT116. The apoptotic effects of TQ are modulated by Bcl-2 protein and are linked to and dependent on p53. Our data support the potential for using the agent TQ for the treatment of colon cancer.
Thymoquinone extracted from black seed triggers apoptotic cell death in human colorectal cancer cells via a p53-dependent mechanism https://pubmed.ncbi.nlm.nih.gov/15375533/
I am quite enthusiastic about Black Seed Oil. It, along with other major players we have discussed, may become part of a regimen that can keep the Spike Protein at bay. If we do all we can to prevent the Spike from entering our cells and limiting/reversing the damage it has caused, we may be able to prevent it from seriously reducing the quality – and length – of our lives. Of course, this is a work of medical research and not medical advice. As always, consult your Primary Care Provider before using any supplement or medication.
Thank you, as always, for your readership, dialogue and support. We added a new Founding Member level paid subscriber this week, and I received a generous donation in the mail. I am humbled and grateful. I will continue working to further our understanding and increase our ability to heal. Please have a blessed Spring weekend! 星期五希望：黑色种子油：抑制尖峰结合，炎症和凝血反应和增强P53表达胸腺酮（TQ），黑色种子油的主要成分，可能是预防/治疗SP的破坏性效果的有用辅助疗法 沃尔特M Chesnut 5月2日在APP替米诺酮中读取可以防止SARS-COV-2病毒受体相互作用，抑制病毒穗糖蛋白与血管紧张素转化酶2（ACE2）的附着 现在正在引起全世界医生的注意力肿瘤抑制器P53的水平令人愉快地下降。 这只是一个示例：遗憾的是，这种时尚的读者知道这已经预期，因为先前讨论了尖刺蛋白与P53和其他肿瘤抑制剂的相互作用。 我在前一周五的希望中写了一些可以增强p53表达的自然治疗方法。 今天，我想讨论一个额外的自然治疗性，不仅可以增强p53表达，而且防止穗状体结合和改善它诱导的炎症/凝血反应。 黑色种子油或黑色孜然油，具有长而高度历史的重要性，多功能的营养保健品。 一个辛辣的药草是Nigella sativa L.（毛茛科），也称为黑孜然或黑色种子，以其烹饪用途而闻名，并且在传统医学中是历史上珍贵的。 黑孜然是东地中海东部地中海，北非，印度次大陆和西南亚的广大地区，在许多国家培育，包括埃及，伊朗，希腊，叙利亚，阿尔巴尼亚，土耳其，S。 在精油，糊状，粉末和提取物的形式是一种灵敏度，黑孜然，在传统医学中表明了许多疾病/条件，如哮喘，支气管炎，风湿病，头痛，背部疼痛，厌食，闭经 这些传统用途的N.苜蓿种子在很大程度上归因于它们各种药用性能，包括抗氧化剂，抗炎，免疫调节，抗癌，神经保护，抗菌剂，抗高血压，卡 黑孜然种子，特别是其精油，含有胸腺醌（TQ），胸腺氢醌，胸腺，碳酸，尼甘油，核苷酸和α-巢蛋白，主要是对其负责的 黑孜然的食物价值虽然在科学文献中较少焦点，但绝不是低的，因为它含有足够的蛋白质和脂肪量，以及可明显量的必需量的必需脂肪酸，氨基酸，维生素和矿物质[7]。 活性植物化学物质和黑孜然的重要营养素同样为人体的免疫和福祉贡献，使这款烹饪草本是一种有价值的营养素来源。 黑雀（Nigella Sativa L.）：植物化学，健康益处，分子药理和安全HTTPS://pmc.ncbi.nlm.nih.gov/articles/pmc8225 黑色种子油及其主要成分胸腺醌（TQ），是Covid和穗蛋白损伤/疾病的有希望的治疗方法。 其中三种益处非常重要：防止尖峰与我们的细胞结合并伸出炎症和凝血反应，即穗和病毒本身诱导。 衍生自N型苜蓿油的化合物，特别是TQ，可有效抑制SARS-COV-2附着于宿主细胞受体并在细胞内复制的能力。 Covid 19还与对SARS-C0V-2病毒的侵袭性炎症和过度活跃的宿主免疫应答有关，该病毒通常被称为“细胞因子风暴”.52促炎细胞因子/趋化因子，包括多个插曲 Nigella sativa的主要组成部分在治疗炎症和传染病HTTPS：//onlinelibrary.wiley.com/doi/10.1111/1440-1681.13/1440-1681.11/1440-1681.11/1440-1681.11/1440-1681 胸腺量具有增强肿瘤抑制P53的表达的能力，该P53的表达是尖刺蛋白下调的。 它还抑制结肠癌细胞的生长。 我们调查了紫醌（TQ）对HCT-116人结肠癌细胞的影响，并试图确定其潜在的分子作用机制。 我们认为TQ抑制结肠癌细胞的生长，所述结肠癌细胞与细胞周期的G1相位阻滞相关。 此外，TUNEL染色和流式细胞术分析表明TQ以剂量和时间依赖的方式触发细胞凋亡。 TQ细胞凋亡诱导与P53和下游P53靶基因MRNA表达的2.5-4.5倍的增加相关。 同时，我们发现P53和P21WAF1蛋白水平显着增加，但对抗凋亡Bcl-2蛋白的显着抑制。 与PIFITHRIN-α（PFT-α）的培养，P53的特异性抑制剂，恢复BCL-2，P53和P21WAF1水平，以未处理的对照和抑制TQ诱导的细胞周期停滞和细胞凋亡 P53-NULL HCT-116细胞对TQ诱导的生长骤停和细胞凋亡不太敏感。 这些结果表明，TQ是抗肿瘤癌细胞系HCT116的抗肿瘤和促凋亡。 TQ的凋亡效应由Bcl-2蛋白调节，并与p53相关联并依赖于p53。 我们的数据支持使用代理TQ治疗结肠癌的可能性。 从黑色种子中提取的胸腺量通过p53依赖机制从黑色种子触发凋亡细胞死亡，通过p53依赖机制https://pubmed.ncbi.nlm.nih.gov/1537533/1537533/ 与我们所讨论的其他主要参与者一起，可能成为可以在海湾保持尖刺蛋白的方案的一部分。 如果我们尽我们所能防止尖峰进入我们的细胞并限制/扭转它造成的损害，我们可能能够防止它严重降低我们生活的质量和长度。 当然，这是医学研究的工作，而不是医学建议。 一如既往，请在使用任何补充或药物之前咨询您的初级保健提供者。 衷心感谢您的读者，对话和支持。 我们本周添加了一个新的创始成员级薪水订阅者，我收到了邮件中的慷慨捐款。 我很谦卑和感激。 我将继续努力进一步了解我们的理解并提高我们的愈合能力。 请有一个幸福的春天周末！
Friday Hope: Black Seed Oil: Inhibiting Spike Binding, Inflammatory and Coagulation Responses and Enhancing p53 Expression
Thymoquinone (TQ), the major component of Black Seed Oil, may be a useful adjunct therapy in preventing/treating the damaging effects of the Spike Protein.
WALTER M CHESNUT
MAY 2
星期五希望：黑色种子油：抑制尖峰结合，炎症和凝结反应并增强p53表达
黑籽油的主要成分胸醌（TQ）可能是防止/治疗峰值蛋白的破坏作用的有用辅助疗法。
Walter M Chesnut
5月2日
https://open.substack.com/pub/wmcresearch/p/friday-hope-black-seed-oil-inhibiting
Friday Hope: Black Seed Oil: Inhibiting Spike Binding, Inflammatory and Coagulation Responses and Enhancing p53 Expression
Thymoquinone (TQ), the major component of Black Seed Oil, may be a useful adjunct therapy in preventing/treating the damaging effects of the Spike Protein.
WALTER M CHESNUT
MAY 2
READ IN APP
Thymoquinone may prevent SARS- CoV- 2 virus receptor interaction by inhibition of the attachment of the viral spike glycoprotein to the Angiotensin Converting Enzyme 2 (ACE2) receptor and the priming of SARS- CoV- 2 virus through TMPRSS2 (transmembrane protease serine 2).
It is now coming to the attention of doctors worldwide that levels of the tumor suppressor p53 are declining alarmingly. Here is just one example:
Unfortunately, readers of this Substack know that this has been expected due to the previously discussed interaction of the Spike Protein with p53 and other tumor suppressors. I have written in a previous Friday Hope about some natural therapeutics which can enhance p53 expression. Today I would like to discuss one additional natural therapeutic which not only enhances p53 expression but also prevents Spike binding and ameliorates the inflammatory/coagulation responses it induces.
Black Seed Oil, or Black Cumin Oil, has a long and highly regarded history of being an important, multi-functional nutraceutical.
One spicy, medicinal herb is Nigella sativa L. (Ranunculaceae), also called black cumin or black seeds, which is famous for its culinary uses, and is historically precious in traditional medicine. Black cumin is native to a vast region of the eastern Mediterranean, northern Africa, the Indian subcontinent, and Southwest Asia, and is cultivated in many countries, including Egypt, Iran, Greece, Syria, Albania, Turkey, Saudi Arabia, India, and Pakistan. Being a panacea, black cumin, in the form of essential oil, paste, powder, and extract, has been indicated in traditional medicine for many diseases/conditions, such as asthma, bronchitis, rheumatism, headache, back pain, anorexia, amenorrhea, paralysis, inflammation, mental debility, eczema, and hypertension, to name a few [4]. These traditional uses of N. sativa seeds are largely attributed to their wide array of medicinal properties, including antioxidant, anti-inflammatory, immunomodulatory, anticancer, neuroprotective, antimicrobial, antihypertensive, cardioprotective, antidiabetic, gastroprotective, and nephroprotective and hepatoprotective properties [5]. Black cumin seed, particularly its essential oil, contains thymoquinone (TQ), thymohydroquinone, thymol, carvacrol, nigellidine, nigellicine, and α-hederin, which are mostly responsible for its pharmacological effects and therapeutic benefits [6]. The food value of black cumin, although less focused on in scientific literature, is by no means low, because it contains an adequate quantity of protein and fat, and an appreciable amount of essential fatty acids, amino acids, vitamins, and minerals [7]. Both active phytochemicals and the vital nutrients of black cumin contribute equally to the immunity and well-being of the human body, making this culinary herb a valuable source of nutraceuticals.
Black Cumin (Nigella sativa L.): A Comprehensive Review on Phytochemistry, Health Benefits, Molecular Pharmacology, and Safety
https://pmc.ncbi.nlm.nih.gov/articles/PMC8225153/
There are additional benefits to Black Seed Oil which are specifically related to SARS-CoV-2 and its Spike Protein. Black Seed Oil and its major component, Thymoquinone (TQ), is a promising therapeutic for COVID and Spike Protein injury/disease. Three of these benefits are very important: preventing the Spike from binding to our cells and quelling the inflammatory and coagulation responses that the Spike and the virus itself induces.
Compounds derived from N. sativa oil, in particular TQ, may be effective in inhibiting the ability of SARS- CoV- 2 to attach to host cell receptors and replicate within the cell.
Covid 19 is also associated with an aggressive inflammatory and hyperactive host immune response to the SARS- C0V- 2 virus which is commonly referred to as a ‘cytokine storm’.52 Proinflammatory cytokines/chemokines, including multiple interleukins and TNF- α, are elevated in Covid19 patients and contribute to worse patient outcomes.52 Nigella sativa and TQ may be potentially beneficial in downregulating this inflammatory process and thereby reducing the severity of acute respiratory distress syndrome.51,52 Nigella sativa and TQ also possess anticoagulant properties that may offer both as potential therapeutic natural products targeting the coagulopathy associated with life- threatening SARS- CoV- 2 (Covid 19) infection.
The role of thymoquinone, a major constituent of Nigella sativa, in the treatment of inflammatory and infectious diseases
https://onlinelibrary.wiley.com/doi/10.1111/1440-1681.13553
Furthermore, there is yet another property that TQ possesses, which may prove to be very beneficial for those exposed to the Spike Protein. Thymoquinone has the ability to enhance expression of the tumor suppressor p53, which the Spike Protein downregulates. It also inhibits the growth of colon cancer cells.
We investigated the effects of thymoquinone (TQ) against HCT-116 human colon cancer cells and attempted to identify its potential molecular mechanisms of action. We report that TQ inhibits the growth of colon cancer cells which was correlated with G1 phase arrest of the cell cycle. Furthermore, TUNEL staining and flow cytometry analysis indicate that TQ triggers apoptosis in a dose- and time-dependent manner. Apoptosis induction by TQ was associated with a 2.5-4.5-fold increase in mRNA expression of p53 and the downstream p53 target gene, p21WAF1. Simultaneously, we found a marked increase in p53 and p21WAF1 protein levels but a significant inhibition of anti-apoptotic Bcl-2 protein. Co-incubation with pifithrin-alpha (PFT-alpha), a specific inhibitor of p53, restored Bcl-2, p53 and p21WAF1 levels to the untreated control and suppressed TQ-induced cell cycle arrest and apoptosis. p53-null HCT-116 cells were less sensitive to TQ-induced growth arrest and apoptosis. These results indicate that TQ is antineoplastic and pro-apoptotic against colon cancer cell line HCT116. The apoptotic effects of TQ are modulated by Bcl-2 protein and are linked to and dependent on p53. Our data support the potential for using the agent TQ for the treatment of colon cancer.
Thymoquinone extracted from black seed triggers apoptotic cell death in human colorectal cancer cells via a p53-dependent mechanism
https://pubmed.ncbi.nlm.nih.gov/15375533/
I am quite enthusiastic about Black Seed Oil. It, along with other major players we have discussed, may become part of a regimen that can keep the Spike Protein at bay. If we do all we can to prevent the Spike from entering our cells and limiting/reversing the damage it has caused, we may be able to prevent it from seriously reducing the quality – and length – of our lives. Of course, this is a work of medical research and not medical advice. As always, consult your Primary Care Provider before using any supplement or medication.
Thank you, as always, for your readership, dialogue and support. We added a new Founding Member level paid subscriber this week, and I received a generous donation in the mail. I am humbled and grateful. I will continue working to further our understanding and increase our ability to heal. Please have a blessed Spring weekend! 星期五希望：黑色种子油：抑制尖峰结合，炎症和凝血反应和增强P53表达胸腺酮（TQ），黑色种子油的主要成分，可能是预防/治疗SP的破坏性效果的有用辅助疗法 沃尔特M Chesnut 5月2日在APP替米诺酮中读取可以防止SARS-COV-2病毒受体相互作用，抑制病毒穗糖蛋白与血管紧张素转化酶2（ACE2）的附着 现在正在引起全世界医生的注意力肿瘤抑制器P53的水平令人愉快地下降。 这只是一个示例：遗憾的是，这种时尚的读者知道这已经预期，因为先前讨论了尖刺蛋白与P53和其他肿瘤抑制剂的相互作用。 我在前一周五的希望中写了一些可以增强p53表达的自然治疗方法。 今天，我想讨论一个额外的自然治疗性，不仅可以增强p53表达，而且防止穗状体结合和改善它诱导的炎症/凝血反应。 黑色种子油或黑色孜然油，具有长而高度历史的重要性，多功能的营养保健品。 一个辛辣的药草是Nigella sativa L.（毛茛科），也称为黑孜然或黑色种子，以其烹饪用途而闻名，并且在传统医学中是历史上珍贵的。 黑孜然是东地中海东部地中海，北非，印度次大陆和西南亚的广大地区，在许多国家培育，包括埃及，伊朗，希腊，叙利亚，阿尔巴尼亚，土耳其，S。 在精油，糊状，粉末和提取物的形式是一种灵敏度，黑孜然，在传统医学中表明了许多疾病/条件，如哮喘，支气管炎，风湿病，头痛，背部疼痛，厌食，闭经 这些传统用途的N.苜蓿种子在很大程度上归因于它们各种药用性能，包括抗氧化剂，抗炎，免疫调节，抗癌，神经保护，抗菌剂，抗高血压，卡 黑孜然种子，特别是其精油，含有胸腺醌（TQ），胸腺氢醌，胸腺，碳酸，尼甘油，核苷酸和α-巢蛋白，主要是对其负责的 黑孜然的食物价值虽然在科学文献中较少焦点，但绝不是低的，因为它含有足够的蛋白质和脂肪量，以及可明显量的必需量的必需脂肪酸，氨基酸，维生素和矿物质[7]。 活性植物化学物质和黑孜然的重要营养素同样为人体的免疫和福祉贡献，使这款烹饪草本是一种有价值的营养素来源。 黑雀（Nigella Sativa L.）：植物化学，健康益处，分子药理和安全HTTPS://pmc.ncbi.nlm.nih.gov/articles/pmc8225 黑色种子油及其主要成分胸腺醌（TQ），是Covid和穗蛋白损伤/疾病的有希望的治疗方法。 其中三种益处非常重要：防止尖峰与我们的细胞结合并伸出炎症和凝血反应，即穗和病毒本身诱导。 衍生自N型苜蓿油的化合物，特别是TQ，可有效抑制SARS-COV-2附着于宿主细胞受体并在细胞内复制的能力。 Covid 19还与对SARS-C0V-2病毒的侵袭性炎症和过度活跃的宿主免疫应答有关，该病毒通常被称为“细胞因子风暴”.52促炎细胞因子/趋化因子，包括多个插曲 Nigella sativa的主要组成部分在治疗炎症和传染病HTTPS：//onlinelibrary.wiley.com/doi/10.1111/1440-1681.13/1440-1681.11/1440-1681.11/1440-1681.11/1440-1681 胸腺量具有增强肿瘤抑制P53的表达的能力，该P53的表达是尖刺蛋白下调的。 它还抑制结肠癌细胞的生长。 我们调查了紫醌（TQ）对HCT-116人结肠癌细胞的影响，并试图确定其潜在的分子作用机制。 我们认为TQ抑制结肠癌细胞的生长，所述结肠癌细胞与细胞周期的G1相位阻滞相关。 此外，TUNEL染色和流式细胞术分析表明TQ以剂量和时间依赖的方式触发细胞凋亡。 TQ细胞凋亡诱导与P53和下游P53靶基因MRNA表达的2.5-4.5倍的增加相关。 同时，我们发现P53和P21WAF1蛋白水平显着增加，但对抗凋亡Bcl-2蛋白的显着抑制。 与PIFITHRIN-α（PFT-α）的培养，P53的特异性抑制剂，恢复BCL-2，P53和P21WAF1水平，以未处理的对照和抑制TQ诱导的细胞周期停滞和细胞凋亡 P53-NULL HCT-116细胞对TQ诱导的生长骤停和细胞凋亡不太敏感。 这些结果表明，TQ是抗肿瘤癌细胞系HCT116的抗肿瘤和促凋亡。 TQ的凋亡效应由Bcl-2蛋白调节，并与p53相关联并依赖于p53。 我们的数据支持使用代理TQ治疗结肠癌的可能性。 从黑色种子中提取的胸腺量通过p53依赖机制从黑色种子触发凋亡细胞死亡，通过p53依赖机制https://pubmed.ncbi.nlm.nih.gov/1537533/1537533/ 与我们所讨论的其他主要参与者一起，可能成为可以在海湾保持尖刺蛋白的方案的一部分。 如果我们尽我们所能防止尖峰进入我们的细胞并限制/扭转它造成的损害，我们可能能够防止它严重降低我们生活的质量和长度。 当然，这是医学研究的工作，而不是医学建议。 一如既往，请在使用任何补充或药物之前咨询您的初级保健提供者。 衷心感谢您的读者，对话和支持。 我们本周添加了一个新的创始成员级薪水订阅者，我收到了邮件中的慷慨捐款。 我很谦卑和感激。 我将继续努力进一步了解我们的理解并提高我们的愈合能力。 请有一个幸福的春天周末！