寄生虫药物对癌细胞有效靶向线粒体底物和糖酵解 相关应用与研究遭政治打压迫害 The application and research on the effective targeting of mitochondrial substrates and glycolysis by anti-parasitic drugs in cancer cells have been politically suppressed and persecuted.
Humanspective @Humanspective "We have a mechanism now why parasite medications are working".
The moment Thomas Seyfried PhD realised how anti parasitic medications work.
@tnseyfried - "Mebendazole, Febendazole [I] did a dive on it and I said, why [do] Parasite medications work against cancer cells, and it turns out that the parasites use mitochondrial substrate level phosphorylation in the tissue .. and mebendazol fenbendazol kill these parasites. So I tried it on the cancer cell and sure as hell, it targets the mitochondrial substrate and glycolysis"
"So we have a mechanism now why parasite medications are working, but cancer is not a parasite"
Interestingly, Thomas Seyfried mentions the political landscape which prevented him from exploring the effects of Ivermectin because of covid, however Dr. Isabella Cooper [ @I_mitochondria ], PhD in Ketogenic Science and Hyperinsulinemia steps in and points out:
"Ivermectin, it actually works on the mitochondrial cell death pathway". 由
NEW ARTICLE: IVERMECTIN and MEBENDAZOLE Testimonial - 7 year old boy in Siberia, Russia, with Cervicomedullary Ganglioglioma reports after 3 months
You never think you're going to be able to help a 7 year old boy in the middle of Siberia, Russia.
STORY: 7 year old boy in Siberia, Russia, with Cervicomedullary Ganglioglioma (a rare brain tumor at the junction of the brainstem and spinal cord, composed of both neuronal and glial cells)
In early August 2025 he started:
Ivermectin 1mg/kg/day Mebendazole 1000mg/day
Result:
“Ivermectin + Mebendazole gave us our son back”
Stable tumor
In Pediatric tumors especially brain tumors, halting tumor growth is a success, as is any clinical improvement. These are very difficult tumors.
Two years ago I never imagined I would be helping over 100 children with cancer.
When Alberta Premier Danielle Smith @ABDanielleSmith sent her entire team of bureaucrats and lawyers to attack 7200+ cancer patients for exercising their "Right to Try" Ivermectin, she was attacking 100+ children as well.
Not a single media outlet has written about these children with cancer who were targeted by Canada's most "conservative" politician.
Article Link in photo to avoid shadowban, just re-type the URL in the first photo at the top, into your browser to access.
Robot Answer: Ivermectin’s action in the body is not limited to its commonly known anti-parasitic and anti-viral functions. It may also affect the mitochondrial cell death pathways, impacting cells on a deeper level.
To clarify this further, we can break it down into two parts: 1. Mechanism of Action of Ivermectin:
Ivermectin is a widely used drug, primarily for treating diseases caused by parasites, such as malaria and river blindness. It works by binding to specific receptors on the cells of parasites, interfering with their nervous system function, and ultimately leading to the death of the parasite. In addition, some studies have found that ivermectin has a certain inhibitory effect on some viruses. 2. Mitochondria and Cell Death Pathways:
Mitochondria are the cell’s powerhouses, responsible for producing ATP (cellular energy). Additionally, mitochondria play an important role in regulating cell death. When mitochondria are damaged, they often initiate apoptosis (programmed cell death) or other forms of cell death. This process is critical in many pathological conditions.
Cell death pathways (such as apoptosis or necrosis) are usually triggered by specific signaling pathways, and sometimes these signals involve changes in mitochondrial function. For example, changes in mitochondrial membrane permeability can lead to leakage of cellular contents, thereby triggering cell death.
Conclusion:
The key point of this sentence is that ivermectin may interfere with the normal functioning of mitochondria through some mechanism, thereby affecting the process of cell death. This effect could extend beyond its traditional role as an anti-parasitic drug, suggesting that it may have potential applications in other diseases (such as certain cancers or viral infections).
The application and research on the effective targeting of mitochondrial substrates and glycolysis by anti-parasitic drugs in cancer cells have been politically suppressed and persecuted.
Humanspective
@Humanspective
"We have a mechanism now why parasite medications are working".
The moment Thomas Seyfried PhD realised how anti parasitic medications work.
@tnseyfried
- "Mebendazole, Febendazole [I] did a dive on it and I said, why [do] Parasite medications work against cancer cells, and it turns out that the parasites use mitochondrial substrate level phosphorylation in the tissue .. and mebendazol fenbendazol kill these parasites. So I tried it on the cancer cell and sure as hell, it targets the mitochondrial substrate and glycolysis"
"So we have a mechanism now why parasite medications are working, but cancer is not a parasite"
Interestingly, Thomas Seyfried mentions the political landscape which prevented him from exploring the effects of Ivermectin because of covid, however Dr. Isabella Cooper [
@I_mitochondria
], PhD in Ketogenic Science and Hyperinsulinemia steps in and points out:
"Ivermectin, it actually works on the mitochondrial cell death pathway".
由
翻译自 英语
“我们现在已经了解了寄生虫药物起作用的机制”。
Thomas Seyfried 博士意识到抗寄生虫药物的作用机制的那一刻。
@tnseyfried
——“甲苯咪唑、芬咪唑[我]深入研究过，我说，为什么[ do]寄生虫药物对癌细胞有效，结果发现寄生虫利用线粒体底物水平磷酸化作用于组织……而甲苯咪唑、芬咪唑可以杀死这些寄生虫。所以我用它们治疗癌细胞，果然，它们靶向线粒体底物和糖酵解。”
“所以我们现在知道了为什么抗寄生虫药物有效，但癌症并不是寄生虫。”
有趣的是，托马斯·塞弗里德提到，由于新冠疫情，政治环境阻碍了他探索伊维菌素的影响，然而，生酮科学和高胰岛素血症博士伊莎贝拉·库珀博士
@I_mitochondria
指出：
“伊维菌素实际上作用于线粒体细胞死亡途径。”
https://x.com/Humanspective/status/1991596484354273493?s=20
William Makis (McGill Medicine)
@MakisMedicine
NEW ARTICLE: IVERMECTIN and MEBENDAZOLE Testimonial - 7 year old boy in Siberia, Russia, with Cervicomedullary Ganglioglioma reports after 3 months
You never think you're going to be able to help a 7 year old boy in the middle of Siberia, Russia.
STORY:
7 year old boy in Siberia, Russia, with Cervicomedullary Ganglioglioma (a rare brain tumor at the junction of the brainstem and spinal cord, composed of both neuronal and glial cells)
In early August 2025 he started:
Ivermectin 1mg/kg/day
Mebendazole 1000mg/day
Result:
“Ivermectin + Mebendazole gave us our son back”
Stable tumor
In Pediatric tumors especially brain tumors, halting tumor growth is a success, as is any clinical improvement. These are very difficult tumors.
Two years ago I never imagined I would be helping over 100 children with cancer.
When Alberta Premier Danielle Smith
@ABDanielleSmith
sent her entire team of bureaucrats and lawyers to attack 7200+ cancer patients for exercising their "Right to Try" Ivermectin, she was attacking 100+ children as well.
Not a single media outlet has written about these children with cancer who were targeted by Canada's most "conservative" politician.
Article Link in photo to avoid shadowban, just re-type the URL in the first photo at the top, into your browser to access.
https://open.substack.com/pub/makisw/p/ivermectin-and-mebendazole-testimonial-40d
@FLSurgeonGen
由
翻译自 英语
最新文章：伊维菌素和甲苯咪唑治疗案例——俄罗斯西伯利亚一名7岁男孩，患有颈髓神经节胶质瘤，治疗3个月后报告
你永远不会想到自己有一天能帮助到身处俄罗斯西伯利亚腹地的一个7岁男孩。
故事：
俄罗斯西伯利亚一名7岁男孩患有颈髓神经节胶质瘤（一种罕见的脑肿瘤，位于脑干和脊髓的交界处，由神经元和胶质细胞组成）。
2025年8月初，他开始了：
伊维菌素 1mg/kg/天
甲苯咪唑 1000 毫克/天
结果：
“伊维菌素+甲苯咪唑让我们找回了儿子”
稳定肿瘤
在儿童肿瘤治疗中，尤其是脑肿瘤，能够阻止肿瘤生长即为成功，任何临床症状的改善也同样如此。这些肿瘤非常难治。
两年前，我从未想过自己会帮助100多名患癌儿童。
当阿尔伯塔省省长丹妮尔·史密斯
@ABDanielleSmith
派出她的整个官僚和律师团队攻击 7200 多名癌症患者行使“尝试使用伊维菌素的权利”时，她也攻击了 100 多名儿童。
没有一家媒体报道过这些身患癌症的儿童，他们成为了加拿大最“保守”政客的攻击目标。
照片中附有文章链接，为避免被屏蔽，只需将顶部第一张照片中的网址重新输入到浏览器中即可访问。
@FLSurgeonGe
Robot Answer: Ivermectin’s action in the body is not limited to its commonly known anti-parasitic and anti-viral functions. It may also affect the mitochondrial cell death pathways, impacting cells on a deeper level.
To clarify this further, we can break it down into two parts:
1. Mechanism of Action of Ivermectin:
Ivermectin is a widely used drug, primarily for treating diseases caused by parasites, such as malaria and river blindness. It works by binding to specific receptors on the cells of parasites, interfering with their nervous system function, and ultimately leading to the death of the parasite. In addition, some studies have found that ivermectin has a certain inhibitory effect on some viruses.
2. Mitochondria and Cell Death Pathways:
Mitochondria are the cell’s powerhouses, responsible for producing ATP (cellular energy). Additionally, mitochondria play an important role in regulating cell death. When mitochondria are damaged, they often initiate apoptosis (programmed cell death) or other forms of cell death. This process is critical in many pathological conditions.
Cell death pathways (such as apoptosis or necrosis) are usually triggered by specific signaling pathways, and sometimes these signals involve changes in mitochondrial function. For example, changes in mitochondrial membrane permeability can lead to leakage of cellular contents, thereby triggering cell death.
Conclusion:
The key point of this sentence is that ivermectin may interfere with the normal functioning of mitochondria through some mechanism, thereby affecting the process of cell death. This effect could extend beyond its traditional role as an anti-parasitic drug, suggesting that it may have potential applications in other diseases (such as certain cancers or viral infections).
机器人解答：伊维菌素（Ivermectin）在体内的作用不仅限于它通常被认为的抗寄生虫和抗病毒功能，它还可能通过影响线粒体的细胞死亡途径，在细胞层面产生影响。
为了更清楚地解释，我们可以分成两个部分：
1. 伊维菌素的作用机制：
伊维菌素是一种广泛应用的药物，主要用于治疗由寄生虫引起的疾病，如疟疾、河盲症等。它通过与寄生虫细胞上的某些受体结合，干扰其神经系统的功能，最终导致寄生虫死亡。除此之外，伊维菌素还被发现在一些研究中对某些病毒有一定的抑制作用。
2. 线粒体和细胞死亡途径：
线粒体是细胞内的能量工厂，负责产生ATP（细胞能量）。此外，线粒体还在调节细胞死亡方面发挥着重要作用。线粒体受到损伤时，通常会启动细胞凋亡（程序性死亡）或其他形式的细胞死亡途径，这一过程在许多病理状态下非常重要。
细胞死亡途径（如凋亡或坏死）通常通过特定的信号传导路径启动，而这些信号有时会涉及线粒体功能的改变。例如，线粒体膜的通透性变化可能导致细胞内的物质泄漏，从而触发细胞死亡。
总结：
这句话的核心意思是：伊维菌素可能通过某种机制干扰线粒体的正常功能，从而影响细胞死亡的进程。这种作用可能超出了传统上对伊维菌素作为抗寄生虫药物的认识，也提示了它可能在其他疾病（比如一些癌症或病毒感染）中具有潜在的作用。