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The results demonstrate that thiabendazole and fenbendazole can target mitochondrial substrates and glycolysis in combating cancer cells


               
2026 Jan 4, 6:31pm   38 views  0 comments

by HANrongli   follow (0)  

甲苯咪唑、芬苯达唑对抗癌细胞,结果证明,它们确实能靶向线粒体底物和糖酵解
The results demonstrate that thiabendazole and fenbendazole can target mitochondrial substrates and glycolysis in combating cancer cells

Humanspective
@Humanspective
World-renowned cancer biologist reveals the stunning and powerful physiological benefits of nutritional ketosis.

But the bombshell comes when he mentions "Ketosis" amplifies how anti parasitic medications work.

Thomas Seyfried PhD: "we have a mechanism why parasite medications are working".

"Once you put the patient [into] nutritional ketosis, it opens up a whole [new] array of drugs that may have [not worked]"

"Once the body is in this new physiological state, things that were thought to be ineffective becomes super effective.... [and Mebendazole, Febendazole] I did a dive on it and I said, why [do] Parasite medications work against cancer cells, and it turns out that the parasites use mitochondrial substrate level phosphorylation in the tissue .. and mebendazol fenbendazol kill these parasites. So I tried it on the cancer cell and sure as hell, it targets the mitochondrial substrate and glycolysis"

PhD in Ketogenic Science and Hyperinsulinemia steps in and also points out that "Ivermectin, it actually works on the mitochondrial cell death pathway".


翻译自 英语
世界知名的癌症生物学家揭示了营养性酮症的惊人而强大的生理益处。

但真正令人震惊的是,他提到“酮症”可以增强抗寄生虫药物的作用机制。

Thomas Seyfried 博士:“我们已经找到了寄生虫药物起作用的机制”。

“一旦患者进入营养性酮症状态,就可以使用一系列之前可能无效的药物。”

“一旦身体进入这种新的生理状态,以前被认为无效的药物就会变得非常有效……[比如甲苯咪唑、芬苯达唑] 我深入研究过,然后我说,为什么[ do]抗寄生虫药物对癌细胞有效,结果发现寄生虫利用线粒体底物水平磷酸化作用来杀灭癌细胞……而甲苯咪唑、芬苯达唑可以杀死这些寄生虫。所以我用它们来对抗癌细胞,结果证明,它们确实能靶向线粒体底物和糖酵解。”

拥有生酮科学和高胰岛素血症博士学位的专家也指出,“伊维菌素实际上作用于线粒体细胞死亡途径”。

https://x.com/Humanspective/status/2007909859082313833?s=20

甲苯咪唑、芬苯达唑对抗癌细胞,结果证明,它们确实能靶向线粒体底物和糖酵解



对话

Humanspective
@Humanspective

🚨 World-renowned cancer biologist reveals the stunning and powerful physiological benefits of nutritional ketosis.

But the bombshell comes when he mentions "Ketosis" amplifies how anti parasitic medications work.

Thomas Seyfried PhD: "we have a mechanism why parasite medications are working".

"Once you put the patient [into] nutritional ketosis, it opens up a whole [new] array of drugs that may have [not worked]"

"Once the body is in this new physiological state, things that were thought to be ineffective becomes super effective.... [and Mebendazole, Febendazole] I did a dive on it and I said, why [do] Parasite medications work against cancer cells, and it turns out that the parasites use mitochondrial substrate level phosphorylation in the tissue .. and mebendazol fenbendazol kill these parasites. So I tried it on the cancer cell and sure as hell, it targets the mitochondrial substrate and glycolysis"

PhD in Ketogenic Science and Hyperinsulinemia steps in and also points out that "Ivermectin, it actually works on the mitochondrial cell death pathway".

翻译自 英语
🚨世界知名的癌症生物学家揭示了营养性酮症的惊人而强大的生理益处。

但真正令人震惊的是,他提到“酮症”可以增强抗寄生虫药物的作用机制。

Thomas Seyfried 博士:“我们已经找到了寄生虫药物起作用的机制”。

“一旦患者进入营养性酮症状态,就可以使用一系列之前可能无效的药物。”

“一旦身体进入这种新的生理状态,以前被认为无效的药物就会变得非常有效……[比如甲苯咪唑、芬苯达唑] 我深入研究过,然后我说,为什么[ do]抗寄生虫药物对癌细胞有效,结果发现寄生虫利用线粒体底物水平磷酸化作用来杀灭癌细胞……而甲苯咪唑、芬苯达唑可以杀死这些寄生虫。所以我用它们来对抗癌细胞,结果证明,它们确实能靶向线粒体底物和糖酵解。”

拥有生酮科学和高胰岛素血症博士学位的专家也指出,“伊维菌素实际上作用于线粒体细胞死亡途径”。

https://x.com/Humanspective/status/2007909859082313833?s=20
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