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The vaccine carriers used by Moderna and Pfizer contain the same spike protein ORF sequence, which can damage endothelial blood vessel cells and promote cancer


               
2025 Oct 6, 11:56pm   53 views  0 comments

by HANrongli   follow (0)  








The vaccine carriers used by Moderna and Pfizer contain the same spike protein ORF sequence, which can damage endothelial blood vessel cells and promote cancer growth. Supplementation with vitamin D3 and essential trace elements for the human body can help resist and alleviate such harm.

Moderna与辉瑞的疫苗载体中含有相同的刺突蛋白ORF序列,破坏内皮细胞血管、促进癌症生长。维生素D3及人体必需微量元素的补充可抵御缓解伤害

Journalist’s Summary: Whether it’s for vaccine detox or daily health maintenance, relying solely on diet is insufficient to meet the optimal standards for human health. Supplementing with vitamins D3, K2, C, B, as well as zinc and magnesium, is essential. As the ancient saying goes: “Prepare for a rainy day; prevent problems before they arise.” Self-protection leads to longevity for the wise!
France: Freedom, Health, Human Rights
Journalist: Han Rongli

記者摘要:無論是疫苗排毒還是日常健康維護,僅僅依靠飲食都不足以達到人類健康的最佳標準。 補充維生素D3、K2、C、B以及鋅和鎂是必不可少的。 正如古語所說:「為雨天做好準備;在問題發生之前預防它們。」 自我保護導致智者長壽!

法國:自由、健康、人權

記者:韓榮利

记者综述:无论是疫苗💉解毒还是日常保健,补充维生素D3、K2、C、B、锌、镁…,完全依靠饮食无法满足人体的最佳健康状态标准!古曰:未雨绸缪、防患未然!自行防护、智者长寿!/ 法国:自由健康人权记者:韩荣利

Catanzaro, et al. Present a Case which Precisely Illustrates my Hypothesis that the Spike Protein Functions as a “Microtumor”
A previously healthy 31-year-old female developed “turbo” bladder cancer post mRNA – the Spike DNA was found in the tumor. Spike also found in cardiac tumors.
WALTER M CHESNUT
OCT 6

https://open.substack.com/pub/wmcresearch/p/catanzaro-et-al-present-a-case-which

READ IN APP

Three and a half years ago, I wrote that the Spike Protein functions as a microtumor. It does this by ripping through the vasculature to create a tumor microenvironment from which cancer may grow.

IT (COVID) IS ONLY A VASCULAR DISEASE INSOMUCH AS THE SPIKE PROTEIN MUST BREAK THROUGH THE VASCULATURE IN ORDER TO INVADE ORGANS. Precisely. The. Same. Mechanism. Cancer. Employs.

Cancer kills by growing into key organs, nerves, or blood vessels and interfering with and impairing their function. It can begin in almost any human cell. Yet, isn’t this EXACTLY what the Spike Protein is doing? It rips through the endothelium to invade organs and impair their function. The much greater danger is that instead of beginning in “any” cell, the spike is attempting to do this in “every” cell.

Cancer disrupts the cellular destruction and renewal pprocess. As a result, new cells become increasingly abnormal, and old cells live when the body should destroy them. Isn’t this what we are observing?

VASCULAR DISEASE AS CAMOUFLAGE: THE SPIKE PROTEIN OF SARS-CoV-2 AS MICROTUMOR
https://wmcresearch.org/vascular-disease-as-camouflage-the-spike-protein-of-sars-cov-2-as-microtumor/

I further explained this process later that year.

The Spike Protein FUNCTIONS AS A “MICROTUMOR”: Instantaneous "Micrometastases": Induction of Tumor Microenvironment: LONG COVID!
WALTER M CHESNUT
·
DECEMBER 22, 2022
The Spike Protein FUNCTIONS AS A “MICROTUMOR”: Instantaneous "Micrometastases": Induction of Tumor Microenvironment: LONG COVID!
Yes, the very first image. The metabolic effects and inflammation of cancer cells (in this case, the Spike Protein causes THE EXACT SAME metabolic reprogramming and inflammation) inducing Cancer Cachexia cause an altered sense of taste and smell. Also, the same effects cause an atrophy of cardiac muscle and an increased energy consumption by the heart.

Read full story
Now we have evidence that this is almost certainly the case. A previously healthy 31-year-old female developed one of the now all-too-common “turbocancers.” Bladder cancer. An exceedingly rare cancer for someone of that age. What was found in her tumor DNA? The Spike Protein. Exactly as I predicted.

Within circulating tumor DNA, a host–vector chimeric read mapped to chr19:55,482,637–55,482,674 (GRCh38), in cytoband 19q13.42, positioned ~367kb downstream of the canonical AAVS1 safe harbor and ~158 kb upstream of ZNF580 at the proximal edge of the zinc-finger (ZNF) gene cluster. This sequence aligned with perfect 20/20 bp identity to a segment (bases …within the Spike open reading frame (ORF) coding region (bases …of the Pfizer BNT162b2 DNA plasmid reference (GenBank accession OR134577.1), despite the patient only receiving Moderna vaccinations. This apparent paradox is best explained by shared Spike ORF sequences within the expression cassette across both vaccine platforms; because Moderna’s proprietary plasmid sequence has not been deposited in NCBI, BLAST defaults to Pfizer’s published reference as the nearest available match.

Genomic Integration and Molecular Dysregulation in Aggressive Stage IV Bladder Cancer Following COVID-19 mRNA Vaccination
https://zenodo.org/records/17122912

Not only was the Spike Protein detected in this patient’s cancer, it was also found in 10 out of 11 cardiac tumors in COVID survivors.

Immunohistochemical study of heart tumors. (A–D) Expression of spike ARS-CoV-2 in myxoma, papillary fibroelastoma, myxofibrosarcoma in the dedifferentiated zone of Grade 3 chondrosarcoma. (E–G) CD68+ macrophages in papillary fibroelastoma, classical myxoma and proliferating myxoma. (H) CD34+ cells in proliferating myxoma; ×200.

Morphological examination revealed the expression of spike SARS-CoV-2 in tumor cells, endothelial cells, and macrophages in 10 out of 11 heart tumors. Conclusion: The detection of SARS-CoV-2 persistence in endothelium and macrophages as well as in tumor cells of benign and malignant cardiac neoplasms, the increase in the number of these tumors, especially cardiac myxomas, after the pandemic by 2023 may indicate a trend toward an increased risk of cardiac neoplasms in COVID-19 patients, which re-quires further research on this issue and a search for new evidence.

High Risk of Heart Tumors after COVID-19
https://pmc.ncbi.nlm.nih.gov/articles/PMC10608002/

What does this mean? I believe we could be looking at a situation where, like HIV, the Spike Protein (perhaps other SARS-CoV-2 proteins, as well?) may need to be “excised” from the body. There is technology being developed which can accomplish this. Of course, the potential ethical dilemmas behind such treatments and their availability are absolutely terrifying.

Scientists say they have successfully eliminated HIV from infected cells, using Nobel Prize-winning Crispr gene-editing technology.

Working like scissors, but at the molecular level, it cuts DNA so “bad” bits can be removed or inactivated.

The hope is to ultimately be able to rid the body entirely of the virus, although much more work is needed to check it would be safe and effective.

Scientists say they can cut HIV out of cells
https://www.bbc.com/news/health-68609297

There is hope. There is much we can do. I will continue to search for therapeutics which can mute oncogenic environments and improve our body’s ability to remove cancer cells. Please have a blessed week.

Thank you, as always, for your continued support, dialog and readership. Great thanks to the three new paid subscribers since Friday and a kind PayPal donation. Each week, between now and Christmas, I am asking if one reader or subscriber would please become a Founding Member of this Substack. Please consider becoming a paid subscriber. I will keep working and reporting back to

Catanzaro 等人展示了一个案例,精准地说明了我的假设:刺突蛋白的功能就像“微型肿瘤”

一名原本健康的31岁女性在接种mRNA疫苗后患上了“极速型”膀胱癌——在她的肿瘤中发现了刺突蛋白的DNA。在心脏肿瘤中也发现了刺突蛋白。

WALTER M CHESNUT
2 025年10月6日

三年半前,我曾写道,刺突蛋白的作用就像一个微型肿瘤。它通过破坏血管,创造一个肿瘤微环境,从而促进癌症生长。

新冠病毒(COVID)之所以表现为血管疾病,仅仅是因为刺突蛋白必须穿透血管系统才能侵入器官。这正是癌症所使用的机制。

癌症通过扩散到关键器官、神经或血管中,从而干扰和破坏其功能,最终致命。它可以从几乎任何一种人体细胞开始。然而,这不正是刺突蛋白正在做的吗?它穿透内皮细胞,侵入器官并破坏其功能。更大的危险在于,癌症可能从“某些”细胞开始,而刺突蛋白似乎想要在“每个”细胞中都这样做。

癌症会干扰细胞的死亡与更新过程。结果就是:新细胞变得越来越异常,老细胞本应被清除却继续存活。我们现在看到的不正是这一过程的体现吗?

把血管病伪装起来:SARS-CoV-2的刺突蛋白如同微型肿瘤

我在那年稍晚进一步解释了这个过程:

刺突蛋白的功能如同“微型肿瘤”:瞬时“微转移”、肿瘤微环境的诱导、长新冠的机制!
2022年12月22日
是的,从第一张图开始。癌细胞(这里由刺突蛋白引起)导致的代谢效应和炎症会引发癌性恶病质(Cachexia),造成味觉和嗅觉的改变。同样的机制也会导致心肌萎缩和心脏能量消耗增加。

现在我们有证据表明,这种假设很可能是正确的。一名原本健康的31岁女性患上了现如今屡见不鲜的“极速癌”——膀胱癌。这种癌症在这个年龄段极为罕见。而在她的肿瘤DNA中发现了什么?**刺突蛋白。**正如我预测的那样。

在其循环肿瘤DNA中,检测到一个宿主-载体嵌合序列,定位在人类19号染色体19q13.42区域,靠近AAVS1安全位点,并与**辉瑞疫苗DNA质粒中的刺突蛋白开放阅读框(ORF)**的一段完全匹配(20/20碱基对)。尽管这位患者只接种了Moderna疫苗,但出现这一矛盾的最佳解释是:**Moderna与辉瑞的疫苗载体中含有相同的刺突蛋白ORF序列。**由于Moderna的专有质粒序列未在NCBI公开,BLAST默认使用辉瑞的公开序列作为最近匹配项。

基因组整合与分子失调:COVID-19 mRNA疫苗后发生的IV期侵袭性膀胱癌

不仅在这位患者的癌症中检测到了刺突蛋白,在新冠康复者的11个心脏肿瘤中,也有10个检测到了刺突蛋白。

心脏肿瘤的免疫组织化学研究。
(A–D)在心房粘液瘤、乳头状纤维弹力瘤、黏液纤维肉瘤,以及三级软骨肉瘤的去分化区域中检测到刺突蛋白的表达;
(E–G)在不同类型的粘液瘤中检测到CD68+巨噬细胞;
(H)在增殖性粘液瘤中检测到CD34+细胞(×200倍显微镜)。

形态学研究表明,在10个心脏肿瘤中(无论是良性还是恶性),在肿瘤细胞、内皮细胞和巨噬细胞中都检测到了SARS-CoV-2刺突蛋白的表达。研究得出结论:新冠疫情后(至2023年)心脏肿瘤(尤其是心房粘液瘤)的数量明显增加,表明新冠患者可能存在更高的心脏肿瘤风险,这一问题需要进一步研究与证据支持。

新冠后心脏肿瘤高风险研究

这意味着什么?我认为,我们可能正在面对一种类似HIV的情况:**刺突蛋白(甚至可能包括其他SARS-CoV-2蛋白)可能需要被“从体内切除”。**目前已有相关技术正在研发。毫无疑问,这类治疗的伦理问题与可及性令人不安。

科学家表示,他们已成功利用诺贝尔奖得主技术CRISPR从感染细胞中清除HIV。

这项技术像分子剪刀一样,可以精准剪断DNA,移除或失活“有害”片段。

希望有朝一日能彻底清除体内的病毒,尽管要确保这种方法的安全性与有效性仍需大量研究。

科学家:我们可以把HIV从细胞中剪除

**仍然有希望。我们可以做很多事情。**我会继续寻找能抑制致癌环境、增强身体清除癌细胞能力的治疗方法。

祝你这一周平安顺利。

感谢你一如既往的支持、交流与阅读。特别感谢自上周五以来三位新订阅者和一位通过PayPal捐款的朋友。从现在起到圣诞节,我每周都会邀请一位读者或订阅者成为Substack的创始会员。欢迎考虑成为付费订阅者。我会继续努力并带来更多报道。
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